骨桥蛋白对急性肺损伤后肺纤维化大鼠肺组织(2)
作者:佚名; 更新时间:2016-02-22
参考文献
[1] Marshall RP, Bellingan G, Webb S, et al. Fibroproliferation occurs earlyin the acute respiratory distress syndrome and impacts on outcome[J].Am J Respir Crit Care Med, 2000, 162(5):1783-1788.
[2] Ma B, Zhou PY, Ni W, et al. Inhibition of activin receptor-like kinase 5induces matrix metallopeptidase 9 expression and aggravateslipopolysaccharide-induced pulmonary injury in mice [J]. Eur Rev MedPharmacol Sci, 2013, 17(8):1051-1059.
[3] 孟红波,房林,卢少毅,等.干扰素-γ对急性胰腺炎大鼠NF-κB 及细胞因子的影响[J].同济大学学报(医学版),2011,32(2):19-23.
[4] Opal SM,DePalo VA.Anti-inflammatory cytokines [J].Chest,2000,117 (4):1162-1172.
[5] 郭振辉,新,毛宝龄,等.核因子-κB 活化在急性肺损伤发病中的作用[J].中华急诊医学杂志,2003,12(9):597-599.
[6] 田莉,王献华,马小兵,等.TNF-α 介导的NF-κB 信号通路在肺纤维化中的作用[J].现代预防医学,2011,2(38):361-363.
[7] 邓丽萍.骨桥蛋白与急性肺损伤[J].西南军医,2011,13(6):1067-1070.